Over the past nine months, the number of US cases of an emerging, multi-drug resistant fungus has ballooned from 7 to more than 122. What’s more, the fungus, Candida auris, seems to be spreading, according to a field report the Centers for Disease Control released Thursday.
We consume all sorts of things before really knowing how they’re going to affect us, including probiotics and dietary supplements. But given how preliminary our understanding of our gut bacteria is, it’s very likely that some supplements can work in direct opposition of others. For instance, vitamin A might kill a bacteria hypothesized to promote childhood growth.
Body odor is a universal human experience. As such, we as a species put a lot of time, money, and effort into finding ways to eliminate unpleasant natural stenches. But most of us put less time, if any, into understanding what actually causes our malodorous condition. But understanding the processes that create b.o. is the first step to creating a less smelly future.
We are starting to see the kind of incurable infection that scientists have warned us about for years. In January, the Centers for Disease Control and Prevention announced that a woman in Nevada had died from an infection resistant to every available antibiotic.
By building a gigantic petri dish, researchers from Harvard Medical School and Technion-Israel Institute of Technology have produced a jaw-dropping visualization showing bacteria as it mutates to become resistant to drugs.
The human nose is packed with bacteria. Some of its inhabitants can sicken us, but yet other nose-dwellers may hold the key to fighting them off. Today, scientists announced the discovery of a new antibiotic produced by bacteria in human noses. Called lugdunin, the compound can combat Methicillin-Resistant Staphylococcus aureus (MRSA). MRSA can cause a potentially life-threatening infection, and is resistant to some antibiotics.
Despite the best efforts to date, a vaccine for HIV remains beyond our reach. It seems every time researchers get close, the virus mutates to stay a step ahead, creating a biological arms race. But a team of scientists at the University of Texas believe they may have found a way to hobble HIV and drastically reduce its virulence. They want to infect susceptible cells with a “good” virus first, one that will effectively immunize them against HIV.